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It is commndd that liv nzym tsts b obtaind pitinitiating thapy with ATOLIP CV and patd as clinically indicatd. Th hav bn a postmakting pots atal and non-atal hpatic ailu in patints taking statins, including atovastatin. I sious liv injuy with clinical symptoms and/hypbiliubinmia jaundic occus duing tatmnt with ATOLIP CV , pomptly intupt thapy. I an altnat tiology is not ound, dnot stat ATOLIP CV.

Th isk myopathy duing tatmnt with statins is incasd with concunt administation ibic acid divativs, lipid-modiying doss niacin, cyclospoin stong CYP3A4 inhibitos (.g., claithomycin, HIV potas inhibitos and itaconazol).

ATOLIP- TABLT might alscaus ctain sid cts. Th common ons amongst thm a constipation, hadach, stomach pain, waknss tc. Howv somtims it might alscaus som sv sid cts lik allgic actions, blood in uin, chst pain, sv back pain, symptoms liv poblms lik yllowing skin ys tc. I ths sv sid cts appa, it is ncssay tsk mdical hlp ight away.

Atovastatin is a slctiv, comptitiv inhibit3-hydoxy-3-mthylglutayl-conzym A (HMG-CoA) ductas, th at-limiting nzym that convts 3-HMG-CoA tmvalonat, a pcusstols, including cholstol. Cholstol and tiglycids (TG)ciculat in th bloodstam as pat lipopotin complxs. With ultacntiugation, ths complxs spaat inthigh-dnsity lipopotin (HDL), intmdiat-dnsity lipopotin (IDL), low-dnsity lipopotin (LDL) and vy-low-dnsity lipopotin (VLDL) actions. TG and cholstol in th liv a incopoatd intVLDL and lasd intth plasma dlivy tpiphal tissus. LDL is omd om VLDL and is catabolizd pimaily though th high-ainity LDL cpto.

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